COVID-19
Coronavirus disease 2019 is a contagious disease caused by severe acute respiratory syndrome coronavirus 2. The first case was identified in Wuhan, China, in December 2019. The disease has since spread worldwide, leading to an ongoing pandemic.Wikipedia
Symptoms:Fever, cough, fatigue, shortness of breath, loss of taste or smell, sometimes no symptoms at all
Complications:Pneumonia, viral sepsis, acute respiratory distress syndrome, kidney failure, cytokine release syndrome, respiratory failure, pulmonary fibrosis, pediatric multisystem inflammatory syndrome, chronic COVID syndrome
Usual onset:2–14 days (typically 5) from infection
Coronavirus Disease (COVID-19)
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Coronavirus Disease 2019 (COVID-19) | CDC
7 Apr Overall US COVID-19 Vaccine Distribution and Administration Update as of Wed, 07 Apr 2021 06:00:00 EST; 7 Apr Vaccine Adverse Event Reporting System (VAERS) 7 Apr COVID-19 Vaccines that Require 2 Shots
Coronavirus (COVID-19) live map tracker from Microsoft Bing
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Symptoms of Coronavirus | CDC
Influenza (Flu) and COVID-19 are both contagious respiratory illnesses, but they are caused by different viruses. COVID-19 is caused by infection with a new coronavirus (called SARS-CoV-2), and flu is caused by infection with influenza viruses. COVID-19 seems to spread more easily than flu and causes more serious illnesses in some people.
COVID-19 - Wikipedia
Coronavirus disease 2019 (COVID-19) is a contagious disease caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). The first case was identified in Wuhan, China, in December 2019. The disease has since spread worldwide, leading to an ongoing pandemic.. Symptoms of COVID-19 are variable, but often include fever, cough, fatigue, breathing difficulties, and loss of smell and taste.
Coronavirus disease 2019 (COVID-19) - Symptoms and causes ...
Infection with the new coronavirus (severe acute respiratory syndrome coronavirus 2, or SARS-CoV-2) causes coronavirus disease 2019 (COVID-19). The virus that causes COVID-19 spreads easily among people, and more continues to be discovered over time about how it spreads.
COVID-19 Map - Johns Hopkins Coronavirus Resource Center
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COVID-19 pandemic - Wikipedia
The COVID-19 pandemic, also known as the coronavirus pandemic, is an ongoing global pandemic of coronavirus disease 2019 (COVID-19) caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). It was first identified in December 2019 in Wuhan, China.The World Health Organization declared the outbreak a Public Health Emergency of International Concern on 30 January 2020, and later a ...
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Coronavirus (COVID-19)
The World Health Organization (WHO) is building a better future for people everywhere. Health lays the foundation for vibrant and productive communities, stronger economies, safer nations and a better world. Our work touches lives around the world every day – often in invisible ways. As the lead health authority within the United Nations (UN) system, we help ensure the safety of the air we breathe, the food we eat, the water we drink and the medicines and vaccines that treat and protect us. The Organization aims to provide every child, woman and man with the best chance to lead a healthier, longer life.
Network scientists identify 40 new drugs to test against COVID-19
Originally Published at News@Northeastern by Roberto Molar Candanosa
Northeastern researchers have identified 40 new potential drugs that could treat COVID-19.
The findings, reported via Northeastern’s Center for Complex Network Science Research on Wednesday, come from a modeling tool for infection dynamics based on network science—complex math, physics, and computing.
The toolset mapped the way proteins within human cells behave after a cell is hijacked with SARS-CoV-2, the new coronavirus that causes the COVID-19 illness.
“We have prioritized many more drugs, but there are 40 that we are actually trying to move forward,” says Albert-László Barabási, Robert Gray Dodge Professor of Network Science and University Distinguished Professor of physics at Northeastern.
Albert-László Barabási, Robert Gray Dodge Professor of Network Science and University Distinguished Professor of physics at Northeastern. Photo by Adam Glanzman/Northeastern University
Barabási’s team is already in discussions with experimental biologists at Harvard to begin testing these drugs in human cell lines.The interactions of different kinds of proteins within our cells play a key role in orchestrating the complex biochemical reactions that control our bodies. That’s why, Barabási says, the best way to understand how viruses spread is to identify the whole set of the molecular interactions within human cells, and the networks those interactions create as proteins interact with genetic material inside the cell.“Virtually every disease that we have spreads through a cellular network,” he says. “The virus has a particularly fast way of doing that because it is invading the cell, in this case with 26 proteins, effectively doing a very fast and effective perturbation on multiple points of the network.”
The model, which Barabási originally developed with a group of researchers in 2012 to study other human viruses, also predicted that SARS-CoV-2 could potentially attack cells in the brain. That finding, the report states, could help explain recent reports that early COVID-19 symptoms include the loss of the sense of smell and taste.
The new list of drugs adds to other potential treatments that researchers have already identified. Barabási’s model identified several of those drugs, as well as others that had not been considered.
“The reason we can do so is because we are not limiting ourselves to drugs that hit those proteins that the virus targets,” Barabási says. “We can also discover drugs that hit in the neighborhood that the virus targets.”
Quickly after entering the body, SARS-CoV-2 hijacks human cells and retools them into virus-replicating machines. The virus relies on the set of protruding proteins for which all other strains of coronaviruses are named—a set of spikes resembling those of a crown. As those viral proteins latch on to the proteins of a healthy cell, they disrupt basic functions within it, and make it churn out millions more versions of the virus.
The first step in modeling viral infection was understanding which proteins SARS-CoV-2 attacks to hijack human cells. Researchers recently reported that there are 332 of those proteins.
Barabási’s toolset modeled the role of the 332 proteins targeted by the coronavirus and predicted other mechanisms those proteins could trigger within the cell to lead to the symptoms of COVID-19. The model focused on drugs that could fight the virus not just by neutralizing its spike proteins as they first targeted human cells, but by disrupting other interactions within human proteins and genes. These treatments target areas within the cell where the virus works, instead of directly targeting the virus, and could influence the way the proteins that SARS-CoV-2 needs to thrive are encoded.“By simply identifying what neighborhood the virus hits, we can identify what are the potential drugs that are hitting in the same neighborhood, and therefore could be effective against the virus,” Barabási says.And, he says, the best drug candidates will probably be those that don’t target the proteins that SARS-CoV-2 initially attacks but work within the same subcellular neighborhood.
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“Very few drugs on the market are directly targeting the disease proteins themselves,” Barabási says. “Simply knowing what proteins the virus targets will not be enough for us to find the drug, we need to understand very well the network’s neighborhoods of those hitting points.”Marinka Zitnik, an assistant professor of biomedical informatics at Harvard Medical School, used machine learning to help Barabási’s group comb through available data on drugs already on the market or in clinical trials that could be repurposed to treat COVID-19. Joseph Loscalzo, Hersey Professor of the Theory and Practice of Medicine at Harvard Medical School, chairman of the Department of Medicine, and physician-in-chief at Brigham and Women’s Hospital, helped the team discard potential drug candidates that could be significantly toxic or impossible to administer intravenously or orally.
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The team’s progress and updates are being released online as soon as they are available, in an effort to help the global scientific community as it works at breakneck speed to solve the COVID-19 crisis. A report with more data and information will be released as a preprint online and submitted to a peer-reviewed journal for publication, Barabási says.
“We are releasing the results as they emerge so that others can immediately build on them and have the research,” he says. “There is such an imminent need to make progress in this area, that we cannot be slaves of our existing academic or business models.”
For media inquiries, please contact Shannon Nargi at s.nargi@northeastern.edu or 617-373-5718.
Last Updated on August 25, 2020